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aspeur lecture tell us about it. x64 anti-virusq: error: missing create-react-app in my react-app when i try to run my newly created react-app, i get this error: missing create-react-app in my react-app here is what i have in my package.json file "scripts": "start": "react-scripts start", "build": "react-scripts build", "test": "react-scripts test", "eject": "react-scripts eject" , i tried npm start and webpack to run my app, but it did not work for me. how do i fix this? a: your package.json is fine, you just need to install it with npm. npm install -g create-react-app then run the command you would normally run your application: create-react-app my-app cd my-app npm start if you have the error "invalidcreatereactapperror", you just need to install the package that provides create-react-app with npm: npm install -g create-react-app@2.0.1 a: you need to run create-react-app my-app and then cd into my-app and type npm start after that, you have successfully created an app ischemic preconditioning augments glycogen metabolism in brain mitochondria through camp-dependent pathways. previous studies in our laboratory have shown that preconditioning of the heart affords protection of a subsequent ischemic episode. we now report that protection of the brain during post-ischemic insults is mediated via the same pathways. a pre- and post-conditioning protocol was used with a variety of time intervals and reperfusion durations. oxygen consumption was monitored after periods of 10, 20, and 30 min of ischemia and after 10 or 20 min of reperfusion. ischemic preconditioning increased recovery of oxygen consumption after a prolonged period of ischemia as assessed at 30 min of post-ischemic reperfusion and in a second study as assessed after 10 min of reperfusion. oxygen consumption was also increased by ischemic preconditioning after 10 min of reperfusion. the beneficial effects of ischemic preconditioning and adenosine were completely eliminated by the a2a adenosine antagonist 8-(p-sulfophenyl)theophylline. the camp-dependent protein kinase inhibitor, h-8, eliminated the beneficial effects of ischemic preconditioning. the phosphodiesterase inhibitor, isobutylmethylxanthine, blocked the beneficial effects of ischemic preconditioning but a nonspecific phosphodiesterase inhibitor, caffeine, did not. these results are consistent with the hypothesis that ischemic preconditioning protects the brain by increasing mitochondrial biogenesis and preservation of energy production during ischemia and reperfusion.we are now two weeks into the regular season, so it’s time to sit down with the standings and examine our starters and bench. here are a few notes and thoughts on what we have seen so far: i’ll be writing a lot of these over the next week and a half. 84d34552a1